I thought this might be right up your alley to discuss @Anthony Castore . I am seeing the topic pick up again about why you should avoid BPC/TB due to possible increase in cancer likelihood. I can't tell if this is all just fear mongering or not. There doesn't appear to be any evidence regarding this outside of referencing a mouse study that was done: ---------------------------------------------------------------------------------------------------------------------------------------------------------- "In most solid tumors studied in mice, including fibrosarcoma, melanoma, non-small cell lung cancer (NSCLC), colon cancer, and glioblastoma, TB4 overexpression promotes tumor growth, metastasis, and angiogenesis." ----------------------------------------------------------------------------------------------------------------------------------------------------------- One person is stating that since BPC up-regulates VEGF, this would be a pathway towards cancer development (below is what they posted). ----------------------------------------------------------------------------------------------------------------------------------------------- VEGF is historically a promoting factor in oncological aspects rather than causative, but this isn't the only concern. VEGF was originally named "vascular permeability factor" for a reason. It opens gaps between endothelial cells, letting plasma proteins and fluid leak into the interstitial space. Off-target stimulation means edema in tissues that don't need increased perfusion. The problem is that VEGF receptors sit on endothelial cells throughout the entire body, not just in the tissue you're trying to help. So if VEGF reaches non-target tissues, several things go wrong. It can produce off-target angiogenesis, meaning new blood vessel growth where you do not want it. That can produce abnormal, fragile, leaky vessels rather than healthy functional ones. VEGF also increases vascular permeability, so tissue can become swollen or edematous. PMID: 35969170, 20400620

The members have spoke and I listened....Most coaches talk about principles. Some share theory. Very few show you exactly what they do themselves. about to change that. I’m opening up my personal playbook, the protocol I run on myself, to show you how I structure my training, nutrition, supplementation, peptides, and recovery strategies to stay at the top of my game. This isn’t a “one-size-fits-all” plan. It’s the real system I use, built from: - Lab data and cellular feedback loops - Peptide science and mitochondrial optimization - Periodized training matched to performance goals - Nutrition timing dialed to physiology, not fads You’ll see the exact tools, dosages, timing, and reasoning I use and how I adjust based on metrics, recovery, and results. If you’ve ever wondered how a coach integrates the science into a living, breathing system… this is your chance to see it in action. Drop a 🔥 below if you want to see the full breakdown of The Coach’s Protocol.I will likely do this as a webinar. Let me know your thoughts who would be interested in seeing this to kick off our monthly case study feature.

52-yr female. I work out 5-6 days a week. I’m lean and well-muscled. I want to accelerate healing as much as I can w/out impacting recovery and increasing recurrence risk. They are going to do an open repair (direct incision with mesh) because I'm lean and have strong abdominals. The surgeon has observed faster recovery and fewer recurrences with this method. Note - it's fat/fascia vs. intestinal protrusion. Planning on: BPC/TB/KPV near stitches; Red light; Bio-regen from BLL; Other suggestions? Will need to order stuff, so there will be a few days' lag on what I can start. Also, if anyone has experience with recovery and training timelines, I would love the information Thank you!!!! Kate

How are we dosing glutathione?

Most people approach supplements the same way they approach a checklist. Energy is low, so they take something for energy. Sleep is off, so they take something for sleep. Inflammation is present, so they take something to reduce inflammation. On the surface, that feels logical. In practice, it’s why so many people stay stuck. Take a common example. Someone uses magnesium for sleep and it works for a week or two, then the effect fades. The assumption is that the dose is wrong or the product isn’t strong enough. Almost never does anyone ask a better question. What changed in the system that made it stop working? The body is not a collection of independent problems waiting to be patched. It is an integrated, adaptive system built around one central priority: managing energy and information flow. Every symptom you experience is an output of that system. Not random. Not isolated. It is a response. When you take a supplement, you are not fixing anything. You are introducing a signal into that system. That signal interacts with cellular pathways, shifts chemistry, and influences how the body allocates resources. Sometimes that produces a desirable outcome. Sometimes it doesn’t. And sometimes it works briefly before the system adapts and you are right back where you started. This is the point where most protocols quietly fail. This is where most people get stuck. They judge supplements based on whether they work instead of asking a more important question. What did this actually do to the system? If you zoom in at the cellular level, the picture becomes clearer. Every cell is constantly managing the movement of electrons through metabolic pathways, essentially how cells generate energy. That flow determines whether energy is produced efficiently or whether stress signals begin to accumulate. Mitochondria sit at the center of this process, integrating fuel availability, oxygen, and signaling inputs to decide how energy gets generated and how the cell responds. When pressure builds at key points in the system, particularly around the electron transport chain, the balance between electron supply and redox capacity begins to shift, and signaling follows. The system moves away from performance and toward protection.