In one sense, the MRI result has remained stable. I was expecting more than stable, but God had other plans. I choose to accept it and feel grateful for what I have. My story: I am a 63-year-old male with longstanding BPH (Benign Prostatic Hyperplasia). 1. July 2024: My PSA kept increasing. The urologist performed a DRE (Digital Rectal Exam), found no suspicious indications, and referred me for an MRI. The result: Suspect nodule, corresponding to PIRADS 4. 2. The urologist said I was a cancer suspect, suggested I do a prostate biopsy. 3. After doing some research, among others, I learned from Dr. Stephen Petteruti in his YouTube videos. This is one of them: Think Twice Before a Prostate Biopsy: The Evidence You Need to Hear. https://youtu.be/6Crij3C1X9E?si=MMiaCD_f7rNP6F1A 4. The possible side effects of a prostate biopsy include bleeding in the urine, semen, or stool, and difficulty urinating. The worst side effect is the risk of spreading the cancer. 5. I have chosen to explore metabolic therapy instead. 6. Fast forward to September 2025, and I did another MRI scan. The result was PIRADS 4, similar to the MRI result 14 months ago, and no sign of metastasis. 7. With the new MRI result, I asked the urologist, given the stable MRI results after 14 months, I asked him about the potential signs that the nodule is not malignant. 8. The urologist said only a biopsy can determine whether it’s malignant or benign. He kept suggesting I do the biopsy. 9. For the second time, I decided to avoid the biopsy because I think the risk of biopsy-related cancer spread is greater than the benefit. I appreciate that some men do not experience the risks I mentioned; good for them, but I just don't want to take that risk. 10. There is an interesting study that showed an inverse correlation between prostate size and prostate cancer incidence. 11. My prostate size is 127cc (considered large according to the urologist). The PSA density (Prostate size vs PSA) values of ≥ 0.20 contribute towards the suspicion of a prostate malignancy. My current PSA density is 0.19.

As we take charge of our own health, we need to avoid making the wrong decisions on the path we've chosen. Dr. Berg's recent video (https://youtu.be/f9PL6o4OLNw?si=H8fzSBL7iaTWM3kc) confused me, I'm comparing different expert opinions: Prof Seyfried: Only glucose and glutamine fuel C Dr. Casey Peavler: Ketone does not fuel C (https://youtu.be/O-k_6ByR63E?si=rWZJySB2B-KRyDst). Fat does not fuel C (https://youtu.be/-JUf69y8j2E?si=E8_4ZdGnc2y58DPw) VS Jane McLelland: glucose, glutamine, and fat fuel C Dr. Berg: glucose, glutamine, Lipids (fat), BCAA, and ketone fuel C While each person's situation is unique, getting the C fuel right is crucial since it's foundational to our action plan. Please share your thoughts. Thank you.

Hi everyone, I noticed some people have commented that they’re struggling to get their GKI below 2 and/or maintain it. So I thought I’d share some tips. I am day 6 into a 14 day fast. This time I am experimenting with a tea fast of matcha, dandelion root and graviola leaves - they all have very promising data indicating evoking of apoptosis in colorectal cancer cells (and other cancer cells too). They may prevent cancer cells using autolytic cell cannibalism to their advantage (whereby they eat surrounding cells in fermentation-driven stress situations) and give this advantage to T-Cells and NK cells to eat the cancer cells instead. Also, they may reduce glutamine’s capacity to be transported and converted to glutamate. Anyway, I have been able to maintain a GKI of 0.3-0.7 when I am in my “pulse” phase with the following strategies: 1. Fasting for 48 hours - sometimes dry, sometimes water-only and sometimes fat-only - they all have produced similar results, but dry fasting is king! 2. Don’t measure ketones and blood glucose within 2 hours of exercising or waking up and be disheartened - your glucose will be naturally higher as it’s the first fuel source drawn on for anaerobic activity (but ketones will eventually be used over time) and due to the “dawn effect” 3. Gluconeogenesis from protein can increase your blood glucose levels, as well as muscle cannibalism during fasting, so drop your protein intake and/or lift weights to prevent this (disregard the short blood glucose increase - this will stop once you’re fully depleted of glycogen). But don’t overdo it - aim to maintain rather than grow because hypertrophy will stimulate mTOR signalling and throw off autophagy. 4. Don’t only rely on ketone supplements and huge amounts of fat - teach your body to use its own fat because it is not just ketones but the metabolic process of ketogenesis that is effective in metabolic therapy. Also, if you eat a lot of fat and take a lot of ketone supplements, this may drive down your blood glucose reading but your insulin could still be high (which we can’t measure regularly and hyperinsulinemia is a driver of tumour growth). 5. If safe to do so, consider fasting to evoke deeper ketosis. For example, in the first day of fasting my GKI was 4.1. On the second day it was 3.2. Then on the third day, it drove down to 0.7 and has been 0.5-0.7 on days 4, 5 and 6. If you are worried about undesired weight loss or feel unwell when fasting, fat fasting is more gentle and won’t block autophagy as fat has zero impact on insulin. This includes bullet proof coffee, e.g. a little butter and MCT oil but NOT cream. 6. Monitor your GKI more closely after you break the fast - you should not go above 2 unless you’re eating too much protein or (like me) have a mindless, carb-addict relapse! 7. Get over the uncomfortable feeling that comes with going from being jacked to skinny as fuck. I totally get it because I used to be so muscular and with each fast I end up looking like I’ve just come out of a labour camp by the end. But at the end of the day, the longer you can go (safely of course and under medical monitoring or at least with a supportive friend or family member) the better your GKI outcome will be.

Interesting read https://www.amandakingnd.com/blog/nitrites-and-nitrates-are-not-bad-for-you

Hey Warriors 👋 One of the best ways we learn here is by sharing insights about what we’ve already tried — and what’s still on our radar. So: ✅ What therapies, supplements, or repurposed drugs have you already tried? ✅ What’s still on your “to explore” list? Please vote and then share in the comments — even a short note helps. Examples could be IV Vitamin C, HBOT, fasting, repurposed drugs, or natural compounds.