Does anyone here have any experience with this? I just purchased it and a 660nm red light.

https://www.facebook.com/share/p/1CU1QfHe5u/ Johns Hopkins’ New Mebendazole Patent and Its Significance for Cancer Therapeutics A recent public news article has highlighted a development that deserves serious attention within oncology and drug-repurposing research: Johns Hopkins scientists have patented a new crystalline form of mebendazole—referred to as polymorph C—designed to enhance its anti-cancer properties. Background: Why Mebendazole Matters Mebendazole is a benzimidazole-class anthelmintic with a well-characterised safety profile accumulated over ~40 years of clinical use. Beyond its antiparasitic activity, numerous preclinical studies have demonstrated: Microtubule inhibition in cancer cells Disruption of glucose metabolism in malignant tissues Interference with multiple signalling pathways (Hh, Wnt/β-catenin, Bcl-2) Selective cytotoxicity to tumour cells at concentrations tolerated by normal cells Despite these properties, clinical adoption has been limited largely because the original molecule is off-patent, making large-scale commercial trials financially unviable. What Johns Hopkins Has Patented The new patent centers on: Polymorph C — a redesigned crystalline form This form appears to demonstrate: Improved oral absorption Higher plasma concentrations Prolonged systemic exposure Greater potency in killing cancer cells in vitro compared with existing polymorphs This is scientifically notable, because mebendazole traditionally suffers from: Poor gastrointestinal absorption High inter-individual variability Low bioavailability unless taken with high-fat meals A more bioavailable crystalline form directly addresses these limitations. Synergy Through Transporter Inhibition The Johns Hopkins team also referenced co-administration with elacridar, a potent inhibitor of: P-glycoprotein (P-gp) Breast Cancer Resistance Protein (BCRP) These efflux pumps are responsible for removing chemotherapeutic agents from cancer cells.

Mebendazole....Anyone using this? Looking for a dose chart please.

Awesome informational paper on the uses of DMSO. https://www.midwesterndoctor.com/p/dmso-mixtures-transform-natural-medicine?r=1aazmf&triedRedirect=true

Hey Warriors, I wanted to share an honest update about my mum’s latest scan and what I’ve learned through this process — I hope it helps others make more informed decisions. Unfortunately, my mum’s recent PET scan results weren’t good. For context, the previous scan in June showed a mixed response with one new lesion appearing. Because of financial constraints, we stopped IV Vitamin C and HBOT about four months ago, and I had planned to strengthen her protocol with additional off-label therapies — Low Dose Naltrexone (LDN), Doxycycline, Hydroxychloroquine, and Niclosamide — all discussed in Jane McLelland’s book How to Starve Cancer. My mum completed the 3-month Orthomolecular protocol back in June and that’s when we saw positive but mixed results. At the time, my mum had strong faith in the NHS oncologists and their plan with immunotherapy (Keytruda). They weren’t sure if the June scan reflected pseudoprogression or inflammation, and even though the radiologist advised a CT scan for clarification, the oncologists decided to wait until the next PET scan. Sadly, that delay cost us valuable time. The latest scan now shows more lesions, and we were told now that SRS/Gamma Knife is no longer an option. The oncologist’s words were: “It’s too late for that.” Also CA125 jumped from 395 to 1100 in 3/4 months. The NHS is an incredible resource in many ways, especially because it funds chemotherapy and immunotherapy, but most NHS doctors have little or no understanding of metabolic therapy. They tend to dismiss it or even speak negatively about it. I printed out research papers and handed them directly to the team, but they ignored them completely. My mum followed their advice because she trusted them, which is understandable, but this experience has changed her perspective. Now she wants to move forward under integrative expert guidance, possibly through Astron Health or a similar precision-medicine approach. From Jane McLelland’s book, I learned that adenocarcinomas (like my mum’s) are generally glutamine-driven, meaning they rely heavily on glutamine as a primary fuel source. We target glutamine in several ways — through fasting and exercise simultaneously, which deplete available fuel, and more importantly through the use of specific off-label drugs. One of the most powerful glutamine antagonists is DON (6-diazo-5-oxo-L-norleucine), though it’s out of reach for most of us due to limited access and high cost. That’s why it’s crucial to investigate or implement other off-labels that can impact glutamine metabolism indirectly. This is also where diet becomes strategic: a pescatarian approach, as Jane suggests, or a vegan keto diet during the kill phase makes a lot of sense for targeting both glutamine and methionine metabolism simultaneously.