You can read about shock all day. You can memorise the four phenotypes, draw the haemodynamic diagrams, recite the SSC bundles. None of that matters if the first time you see a septic patient transition from warm to cold shock is on a real transport at two in the morning with a 90-minute flight ahead of you. That is what simulation is for. Not to test you. To let you fail safely, recognise the patterns, and build the clinical reasoning that works under pressure. We have built a complete simulation pack for the Shock Module. Five scenarios. Each one comes in two formats: a Relaiti 360 / iSimulate scenario file (.isims) that you can import directly into the app and run as a live vital signs simulation, and a tabletop discussion document (.docx) designed to be worked through as a group with a facilitator. The five scenarios: SHOCK001 — Septic Shock: The Warm-to-Cold Transition. A 72-year-old with urosepsis who starts as textbook warm shock and then transitions to cold shock mid-scenario. The group has to recognise that this is myocardial depression, not fluid overload, and adjust their vasopressor strategy accordingly. The metformin on the medication list forces a discussion about lactate interpretation. SHOCK002 — Cardiogenic Shock: The Post-STEMI Fluid Trap. A 58-year-old anterior STEMI where the sending ED has given a litre of fluid for the low blood pressure. The patient now has elevated JVP, bilateral crackles, and worsening hypotension. The group has to identify that the fluid was the wrong treatment, withhold further volume, and manage the oxygenation spiral from iatrogenic pulmonary oedema. SHOCK003 — Obstructive Shock: Tension Pneumothorax in the Ventilated Patient. A 42-year-old trauma patient, intubated, ICC in situ, stable on departure. Twenty minutes into a fixed-wing transport the ventilator starts alarming. The group works through DOPES, identifies the tension pneumothorax clinically without imaging, and decompresses. The teaching point: positive pressure ventilation converted a simple pneumothorax to tension through a blocked ICC.

You are transporting a patient with anaphylactic shock who has received three doses of IM adrenaline 0.5 mg with minimal response. Their MAP is 38 mmHg. Using receptor pharmacology and pharmacokinetics, explain at least three reasons why IM epinephrine may be failing, and describe your complete escalation strategy including route, dose, preparation, adjuncts, and monitoring. To refresh your knowledge on this chekc out the vasopressor pharmacology section out.

Hey all. Have juts put the finishing touches on the written content for the above course material . Juts going through adding images and refinining it further. Feel free to comment on any improvements

A quick refresher on Ventilator settings...