In yesterday’s lesson, we broke down how insulin moves glucose into your muscle, liver, and fat. Today, we connect the dots — how chronically high insulin flips that system upside down. When your muscles stop listening to insulin, your liver is forced to convert glucose into fat (de novo lipogenesis). It packages that fat into VLDL particles and ships them into the bloodstream. Then insulin activates LPL in your fat tissue, which pulls that fat straight out of the blood and locks it into storage. At the same time, insulin resistance and inflammation in your brain shut down kisspeptin neurons, lowering GnRH, LH, and ultimately testosterone. This is the full picture of modern metabolic collapse — and the reason most men are tired, soft, and low-T despite “normal” labs. 🧩 You’ll Learn: - How the liver converts excess glucose into fat (VLDL pathway) - Why LPL stays “on” in fat cells during hyperinsulinemia - How brain insulin resistance blunts GnRH and testosterone - The top 3 ways to start reversing it today (fasting windows, post-meal movement, and resistance training) 💥 Takeaway: High insulin starts in your blood — but ends in your brain. Fix the signal, and everything downstream begins to work again. Comment your biggest takeaway below — what’s the first lever you’re pulling to fix insulin and restore your hormones? #PerformanceMedicine #MensHealth #Metabolism #CovenixBrotherhood