AI Analysis Of My Mum’s Protocol
There is a new feature on ChatGPT called “Deep Research”. I ran my mum’s protocol using this tool. It took about 15 minutes. Here’s the result: Regimen Overview and Treatment Goals Supplement/Medication List: The user’s regimen includes antiparasitic medications (ivermectin, mebendazole, fenbendazole, artemisinin), anti-inflammatory/antioxidant supplements (curcumin, quercetin, resveratrol, EGCG from green tea, vitamin E), vitamins/minerals (vitamin D3, vitamin C via camu camu, zinc, magnesium, omega-3 fish oil), a liver support herb (milk thistle), a metabolic supplement (berberine), and others like creatine. Additionally, the user is on rivaroxaban (Xarelto), a blood-thinning medication. This combination strongly suggests an integrative therapy aimed at cancer treatment or a complex chronic condition – leveraging repurposed drugs and supplements to inhibit tumor growth, support the immune system, and protect organ function. Below we analyze how each component fits the likely treatment goals, their mechanisms of action, and how they might interact with one another. Mechanisms of Action and Relevance to Goals • Antiparasitic Drugs with Anticancer Properties: Ivermectin, Mebendazole, Fenbendazole, Artemisinin. Originally anti-parasitic, these have shown broad anti-tumor effects. Ivermectin can inhibit cancer cell proliferation and metastasis by disrupting pathways like Wnt/β-catenin and AKT signaling. It’s been reported to significantly reduce cancer cell motility and metastasis in models. Mebendazole and fenbendazole are benzimidazole compounds that bind to β-tubulin in parasites; in cancer cells they similarly disrupt microtubule formation, impairing cell division. This can induce cancer cell death and has made mebendazole a promising repurposed drug in oncology. Artemisinin, derived from wormwood, is unique: when it encounters high iron levels, it produces reactive free radicals that destroy cells. Cancer cells, which hoard iron for rapid DNA replication, become selective targets – artemisinin can trigger a burst of free radicals that selectively kill iron-rich tumor cells while sparing normal cells. This makes artemisinin a potent pro-oxidant therapy in cancer. In summary, these drugs attack cancer on multiple fronts – inhibiting cell division (mebendazole/fenbendazole), inducing oxidative stress in cancer cells (artemisinin), and blocking pro-survival signaling and metastasis (ivermectin).